Dysplasia : Causes, Types and Treatment Options.

Intro -- DYSPLASIACAUSES, TYPES AND TREATMENT OPTIONS -- Library of Congress Cataloging-in-Publication Data -- CONTENTS -- PREFACE -- CHAPTER 1. COLONIC POLYPS AND HEREDITARY POLYPOSIS SYNDROMES -- ABSTRACT -- INTRODUCTION -- EPIDEMIOLOGY -- RISK FACTORS -- DIET -- PERSONAL HISTORY -- FAMILY HISTORY -- HEREDITARY POLYPOSIS SYNDROMES -- INHERITED ADENOMATOUS POLYPOSIS SYNDROMES -- OTHER INHERITED ADENOMATOUS SYNDROMES -- Familial Adenomatous Polyposis (FAP) -- Genetics -- Clinical Features -- Figures Demonstrating FAP -- Extracolonic Involvement in FAP -- Osteomas -- Gastric and Duodenal Adenomas -- Desmoid Tumors -- Other Tumors -- Gardner's Syndrome (A FAP Variant) -- Genetics -- Clinical Features -- Turcot's Syndrome -- Genetics -- Clinical Features -- Muir's or Torre's Syndrome -- Genetics -- Clinical features -- INHERITED HAMARTOMATOUS POLYPOSIS SYNDROMES -- Peutz-Jegher's Syndrome -- Genetics -- Clinical Features -- Juvenile Polyposis -- Genetics -- Clinical Features -- HEREDITARY NONPOLPOSIS COLORECTAL CARCINOMA -- Genetics -- Clinical Features -- CONDITIONS ASSOCIATED WITH ADENOMATOUS POLYPS -- Acromegaly -- Streptococcus Bovis Bacteremia -- Ureterosigmoidostomy -- Atherosclerosis and Cholesterol -- Cholecystectomy -- DIAGNOSIS AND SCREENING -- Diagnosis -- Signs And Symptoms -- Investigation -- Fecal Occult Blood Testing (FOBT) -- Sigmoidoscopy -- Barium Enema -- Colonscopy -- Endoscope-Compatible Optic Fiber Systems -- Screening -- Familial Polyposis and HNPCC Screening -- Family History of Colorectal Cancer -- Prior Adenoma or Colorectal Cancer -- MANAGEMENT -- Medical Treatment -- Endoscopic Resection -- Resectional Surgery -- REFERENCES -- CHAPTER 2. DEVELOPMENTAL DYSPLASIA OF HIP -- ABSTRACT -- INTRODUCTION -- HIP DEVELOPMENT -- EPIDEMIOLOGY -- CLINICAL PRESENTATION -- IMAGING -- SCREENING -- MANAGEMENT -- COMPLICATIONS -- CONCLUSION.

REFERENCES -- CHAPTER 3. BONE DYSPLASIA: CAUSES, CLASSIFICATION AND TREATMENT OPTIONS -- ABSTRACT -- CLASSIFICATION OF BONE DYSPLASIAS -- TREATMENT AND MANAGEMENT OF SKELETAL DYSPLASIA -- REFERENCES -- CHAPTER 4. DOES ELEVATED INTRACELLULAR CHLORIDE CAUSE EPILEPSY IN FOCAL CORTICAL DYSPLASIA? -- ABSTRACT -- INTRODUCTION -- GABA CAN BE BOTHINHIBITORY ANDEXCITATORY -- GABAERGIC ACTION IS DEPOLARIZING IN FCD -- CHLORIDE COTRANSPORTERS IN HUMAN FOCAL CORTICAL DYSPLASIA -- CHLORIDE HOMEOSTASIS IN ANIMAL MODELS OF EPILEPSY AND CHLORIDECOTRANSPORTER KNOCK-OUT MICE -- POSSIBLE THERAPIES FOR EPILEPSIES INDUCED BY ELEVATED INTRACELLULAR CHLORIDE -- REFERENCES -- CHAPTER 5. DYSPLASIA IN LONGSTANDING ULCERATIVE COLITIS: DIAGNOSIS, TYPES, SURVEILLANCE AND TREATMENT OPTIONS -- ABSTRACT -- INTRODUCTION -- DYSPLASIA IN ULCERATIVE COLITIS -- What Is Dysplasia? -- Why Looking for Dysplasia in UC? -- Because of the Risk of Developing Colonic Cancer! -- So, Who Should We Screen? -- How Should We Diagnose and Screen? -- New Methods for Colon Surveillance -- Once Found, What to Do With It? -- Treatment -- CONCLUSION -- REFERENCES -- CHAPTER 6. CHARACTERIZATION OF ANGIOGENIC ACTIVITY AND IDENTIFICATION OF MEDIATORS IN BRONCHIAL DYSPLASIA AND INVASIVE LUNG CANCER: ROLE OF VASCULAR ENDOTHELIAL GROWTH FACTOR AND ANGIOPOIETINS -- ABSTRACT -- INTRODUCTION -- MATERIALS AND METHODS -- Subjects and Specimens -- Multiplex, Semi-Quantitative PCR Analysis of Ang1, Ang2 and Tie-2 Expression -- In Vitro Characterization of Angiogenic Activity -- Statistical Analysis -- RESULTS -- Subject Population -- Expression of Angiopoietin 1 and 2 and Their Receptor, Tie2 in Bronchial Dysplasia and Invasive Non-Small Cell Lung Carcinomas -- Combined Analysis of VEGF and Angiopoietin Expression in Relation to Angiogenic Activity in Bronchial Dysplasia and Invasive Lung Carcinoma.

In Vitro Characterization of Dysplasia and Tumor Derived Angiogenic Activity: Mechanistic Role of VEGF -- DISCUSSION -- REFERENCES -- CHAPTER 7. SUDDEN CARDIAC DEATH SYNDROME - ARRHYTHMOGENIC RIGHT VENTRICULAR DYSPLASIA/CARDIOMYOPATHY AS MOST FREQUENT CAUSE OF FATAL ARRHYTHMIAS -- ABSTRACT -- Background -- Incidence -- Circadyal Rhythm -- Age -- ETIOLOGY: GENETICS, MUTATIONS, DESMOSOMAL CYTOSOLIC CHANGES -- APOPTOSIS IN THE HEART -- PATHOANATOMICAL AND PATHOPHISIOLOGICAL BASIS OF THE DISEASE -- DEFINITION -- Clinical Forms and Genetics. Classification of Right Ventricular Cardiomyopathies -- ARVD -- Biventricular Dysplasia -- RVD without Arrhythmia Quiescent -- RVD with Congestive Heart Failure -- ARVD + Superimposed Myocarditis -- Quiescent -- Hyper Acute -- Acute -- Chronic -- Chronic-active -- Naxos Disease -- Israelian Desmoplakin Recessive Dysplasia -- ARVD Mimicking Uhl's Anomaly -- Biventricular Spongy Dysplasia -- Catecholaminergic VTs -- Brugada Syndrome (BS) (Some Patients Only) -- Right Ventricular Outflow Tract (RVOT) (about 50% of Patients) -- Septal Ventricular Outflow Tract (SVOT) -- Left Ventricular Outflow Tract (LVOT) -- Fat Dissociation Syndrome (FDS) -- CLINICAL PICTURE -- DIAGNOSIS -- PITFALLS IN DIAGNOSING ARVD -- RELEVANCE OF RIGHT VENTRICULAR FUNCTION -- ECHOCARDIOGRAPHY IN DIAGNOSING ARVD -- DIMENSIONS OF RIGHT VENTRICLE REGION OF INTEREST -- ECG NEWLY OBSERVED SIGNS -- VECTORCARDIOGRAPHY IN DIAGNOSING ARVD -- DISCUSSION -- CONCLUSION -- REFERENCES -- INDEX.

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