Karyogamic Theory of Cancer Cell Formation from the View of the XXI Century.

Intro -- Library of Congress Cataloging-in-Publication Data -- Contents -- Preface -- Chapter 1 -- History of the Problem -- Conclusion -- References -- Chapter 2 -- Hybrid Cells Receiving Methods and Mechanism of Cells' Fusion -- Abstract -- Introduction -- 2.1. Methods of Artificial Fusion of Somatic Cells -- 2.2. Mechanism of Somatic Cells' Fusion -- Conclusion -- References -- Chapter 3 -- Geno- and Phenotypic Characteristics of Hybrid Cells -- Abstract -- Introduction -- 3.1. Cytogenetic Analysis of Hybrid Cells -- 3.2. Morphology of Hybrid Cells -- Conclusion -- References -- Chapter 4 -- Utilization of Somatic Hybridization in Biology and Medicine -- Abstract -- Introduction -- 4.1. Interspecific Hybrids -- 4.2. Intraspecific Hybrids -- 4.3. Production of Monoclonal Antibodies -- Conclusion -- References -- Chapter 5 -- Etiology and Pathogenesis of Cancer -- Abstract -- Introduction -- 5.1. Theories and Hypotheses of Carcinogenesis -- 5.2. Hybridization Theories of Tumorous cells Formation -- Conclusion -- References -- Chapter 6 -- Factors, Induced the Malignant Process -- Abstract -- Introduction -- Conclusion -- References -- Chapter 7 -- The Theory of "Two Synkaryons" -- Abstract -- Introduction -- 7.1. Formation of Precancerous Cell or Synkaryon of Stage I -- 7.2. Arising of the True Tumorous Cell or Synkaryon of Stage II -- 7.3. Molecular-Biological Mechanism of Malignant Transformation -- 7.4. Common Characteristics of a Tumorous Synkaryon -- 7.5. Possible Mechanism of Tumors' Progression -- Conclusion -- References -- Chapter 8 -- Experimental and Clinical Confirmations of the "Two Synkaryons" Theory -- Abstract -- Introduction -- 8.1. Parallelism between Fusogenic and Carcinogenic Abilities of Some Agents and Factors -- 8.2. Parallelism between Polykaryocytosis and Some Histogenetic Forms of Cancer.

8.3. Short-Term Test on Carcinogenicity in the Basis of the Theory of "Two Synkaryons" -- 8.4. Experimental Facts Confirming the Theory of "Two Synkaryons" -- 8.5. Indirect Data, Confirming the Theory of "Two Synkaryons" -- Conclusion -- References -- Chapter 9 -- Explanation of Different Complicated Aspects of the Experimental and Clinical Oncology from the Standpoint of the "Two Synkaryons" Theory -- Abstract -- Introduction -- 9.1. Multi-Step of Carcinogenesis -- 9.2. Spontaneous Malignization In Vitro -- 9.3. Induction of Malignant Tumors in Allotransplantations -- 9.4. Induction of Malignant Tumors in Autoimmune Diseases -- 9.5. Possible Cellular Mechanism of Malignization in Hemolytic Anemias of Different Origin -- 9.6. Macrophages as Initiators of the Neoplastic Process -- 9.7. Possible Mechanism of Malignization by Different "Noncarcinogenic" Agents -- 9.7.1. Induction of Malignant Tumors by Some Fatty Acids -- 9.7.2. Possible Reason for Induction of Malignant Tumors by Some Carbohydrates -- 9.7.3. Induction of Malignant Tumors by Distilled Water -- 9.7.4. Induction of Malignant Neoplasms by Physiological Solution (NaCl) -- 9.8. Foreign-Body Carcinogenesis from the Position of the "Two Synkaryons" Theory -- Conclusion -- References -- Chapter 10 -- Etiology and Pathogenesis of AIDS -- Abstract -- Introduction -- A Possible Reason of Immunocompetent Cells Reduction -- Conclusion -- References -- Chapter 11 -- Possible Common Etiology and Pathogenesis of Cancer and AIDS -- Abstract -- Introduction -- Reason for Simultaneous Development of Cancer and AIDS -- Conclusion -- References -- Chapter 12 -- A Possible Reason for the Interrelation of Some Malignant Tumors and AIDS -- Abstract -- Introduction -- A Possible Reason for Kaposhi's Sarcoma Interrelation with AIDS -- Conclusion -- References -- Chapter 13.

A Paradoxical Idea of Cancer's Resolution -- Abstract -- Introduction -- Perspectives on Cancer's Resolution and Prevention -- Conclusion -- References -- Summary -- References -- Index -- Blank Page.

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Electronic reproduction. Ann Arbor, Michigan : ProQuest Ebook Central, 2024. Available via World Wide Web. Access may be limited to ProQuest Ebook Central affiliated libraries.